RYAN WHITE PART A PRESCRIPTION DRUG FORMULARY Sorted by HRSA d-code ; Revised: 10 12 2007 This is a comprehensive list of medications that may be required by individuals who have HIV or AIDS. All items will be reimbursed in their generic equivalent. Reimbursement for name brand items will only be permitted in the event that a generic equivalent is not available on the market. There may be special situations where medications are needed that are not on this list i.e., HIV-related heart disease or HIV-related kidney failure ; and a mechanism should be set up to deal with such extenuating circumstances. NOTES: * HRSA d-codes are now included as derived from the Multum Lexicon database from Cerner Multum, Inc. This database was modified to fit the Ryan White Prescription Drug Formulary format. A complete copy of the database is available upon request from OSBM. * Medications assigned a letter notation will be provided by Ryan White Part A only if the specified criteria under the designated letter is met. Refer to the end of the formulary for more detail on each letter notation. Drug Classification Protease Inhibitors Nutritional Supplements Ophthalmic Otic Preparations Ophthalmic Otic Preparations Protease Inhibitors Protease Inhibitors Ophthalmic Otic Preparations Psychotropic Medications Antiretroviral Agents Ophthalmic Otic Preparations Ophthalmic Otic Preparations Psychotherapeutics Ophthalmic Otic Preparations Hyperlipidemia Invirase Lactaid Garamycin Ocuflox ophthalmic ; Norvir Crixivan Xalatan Remeron Viramune Timoptic Alphagan Zyrpexa Cortisporin Ophthalmic Lipitor Brand Name Saquinavir Lactase Enzyme Oral ; Gentamicin Ophthalmic Solution & Ointment ; Ofloxacin ophthalmic ; Ritonavir Indinavir Latanoprost ophthalmic Mirtazapine Nevirapine Timolol ophthalmic Brimonidine Olanzapine Hydrocortisone Neomycin Polymyxin B Zinc Solution, Suspension for eyes Atorvastatin. Surgical wounds, infection of, 441. Suture, barbed, for flexor tendon repair, * 578. Synovectomy chemical, experimental study, 435. for rheumatoid arthritis symposium ; , 219. Synovial cysts, intraosseous. See Bone cysts, chondral. Synovioma, malignant, of tendo calcaneus, 676. Synovitis pigmented villonodular bone lesions in, * 306. diagnosis and treatment, * 290. of wrist with penetration into bone, * 3 12. Syringomyelia, scoliosis in, 436 and prozac.
Procedure for the analysis of each of the antidepressant drug pairs. All the tertiary amine tricyclics are well resolved from their secondary amine metabolites and concentrations of each of the antidepressant drug pairs may be determined simultaneously, without derivatization. Interferences between different tricyclics preclude the use of this method for toxicological analysis.
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28.7 kilograms per meter squared kg m 2 Four months later, buspirone was added to his treatment. Within 2 months, his BMI had increased to 34.1 kg m 2 ; Three months later, he had experienced weight loss BMI 27.5 kg m 2 and developed polyuria and polydipsia. Fasting blood glucose was 368 milligrams deciliter. Olanzapine was discontinued. Without hypoglycemic drugs, insulin treatment, or dietary changes, his serum glucose level normalized over the next 8 weeks, as did his serum triglyceride and cholesterol levels. Twenty weeks after the discontinuation of olanzapine, his BMI was 26.5 kg m 2 ; Domon & Webber, 2001 ; . 3.3.3.E Hypoglycemia 1 ; Summary a ; HYPOGLYCEMIC COMA was reported in a frail, 95-year-old woman following olanzapine administration for the treatment of nocturnal psychomotor agitation associated with Alzheimer's dementia. Three days after the initiation of olanzapine at a dose of 2.5 milligrams daily, the woman was pale and sweaty; she appeared sleepy and could not be woken by verbal or tactile stimulation. She was treated with 33% glucose and recovered within approximately 20 minutes, however, hypoglycemia was noted again the following day. Olanzapine was withdrawn and the blood glucose profile stabilized two days later with the ongoing administration of 33% glucose. A direct cause and effect correlation could not be established because the patient had also been receiving enalapril, which has been documented to possibly induce hypoglycemia. While, a drug interaction between enalapril and olanzapine could not be ruled out, the authors did not feel that there was a correlation between enalapril and hypoglycemia because the patient had been receiving enalapril for at least two years previous to this incident Landi et al, 2003 ; . 3.3.3.F Metabolic finding 1 ; Summary a ; The manufacturer reports that INCREASED APPETITE 3-6% ; and WEIGHT GAIN 5-6% ; have occurred with olanzapine therapy. Prod Info Zyprexa R ; , Zyprexa R ; Zydis R ; , Zyprexa R ; IntraMuscular Olanzapine, 2004a ; . 2 ; Weight gain, increased appetite, increases in serum triglycerides, and hyperglycemia have been reported with olanzapine administration. 3 ; Olanzapine-induced KETOACIDOSIS has been reported, including one near fatal case in a 44-year-old African American woman. In the near fatal case, the patient had taken olanzapine 25 milligrams day for approximately one month Straker et al, 2002 ; . 4 ; High dose olanzapine was associated with mild extrapyramidal symptoms EPS ; , elevated serum prolactin and alanine aminotransferase ALT ; levels, and an average weight gain of 8 kilograms kg ; in 8 men with schizophrenia and schizoaffective disorder. These patients, who were resistant to typical neuroleptics and risperidone or clozapine at adequate doses, were dosed with olanzapine 20 to 40 milligrams mg ; for a mean of 40 weeks Bronson & Lindenmayer, 2000 ; . 5 ; A prospective, multicenter, observational study showed that olanzapine treatment of outpatients n 2128 ; with schizophrenia was safer than in a control group of patients n 821 ; receiving a variety of other antipsychotic drug therapies. Drugs used in the control group included risperidone, haloperidol, sertindole, zuclopenthixol, fluphenazine, thioridazine, perphenazine, pimozide, clozapine, pipotiazine, sulpiride, chlorpromazine, levomepromazine, clothiapine, and lorazepam. Overall, olanzapine had a significantly lower incidence of adverse events than the control group 48% versus 64%, p less than 0.001 ; . Somnolence and weight gain occurred significantly more frequently in olanzapine-treated patients. Akathisia, dystonia, extrapyramidal syndrome, hypertonia, and tremor were significantly higher in the control group. Abnormal ejaculation and impotence occurred significantly more frequently in men in the control group. Over a 6-month period, fewer olanzapine-treated patients received a concomitant anticholinergic medication in comparison to patients in the control group 36% versus 58%, p less than 0.001 ; Gomez et al, 2000 ; . 6 ; LITERATURE REPORTS a ; Adolescent patients taking olanzapine experienced greater weight gain and increased in body mass index BMI ; than patients taking quetiapine in a retrospective study involving 103 patients younger than 18 years of age. Patients received olanzapine n 50, mean daily dose 13.9 milligrams mg or quetiapine n 53, mean daily dose 510.9 mg ; for at least 2 weeks. Weight and height were measured at baseline and 14 or more days after baseline. Average weight gain from baseline in the olanzapine group was 3.8 kilograms kg ; p less than 0.001 ; compared to 0.03 kg in the quetiapine group. Both the olanzapine and quetiapine groups showed slight, but significant, increases in height from baseline 0.006 meters, p 0.042 and 0.006 meters, p less than 0.001, respectively ; . After controlling for baseline differences, the mean weight change between groups was significant 3.4 kg, p less than 0.001 ; . BMI increased by an average of 1.3 kg per square meter m 2 in the olanzapine group p less than 0.001 ; compared to a decreased of 0.2 kg m 2 ; the quetiapine group. After controlling for baseline differences, the mean difference in change in BMI was significant 0.9 kg m 2 ; , 0.008 ; Patel et al, 2004 ; . b ; In continuing day-treatment program 15 out of 16 patients receiving olanzapine gained weight. The mean weight gain was 22 pounds with a mean olanzapine dose of 14 milligrams mg ; and mean treatment duration of 7 months Gupta et al, 1999 ; . c ; Weight gain has been observed in recipients of olanzapine mean of 3.5 kilograms kg ; with 12.5 to 17.5 milligrams mg ; daily ; Beasley et al, 1996 ; and is most pronounced in patients on initial doses of 15 mg or above Prod Info Zyprexa R ; , 1998 ; . d ; EXCESSIVE APPETITE was seen more commonly with olanzapine therapy than with haloperidol 24% versus 12.4%, p less than 0.05 ; . Olanzapine therapy was also associated with a clinically significant greater increase in weight over haloperidol therapy p less than 0.001 ; . However, a post hoc analysis revealed that body mass index was the predominant predictor of weight gain. Patients with a low prestudy body mass index were more likely to gain weight during olanzapine treatment. Treatment effect on weight change was consistent between male and female patients Tollefson et al, 1997a ; . 3.3.3.G Prolactin level raised 1 ; Summary a ; A case a GALACTORRHEA with elevated serum prolactin levels was reported in a 33-year-old woman after receiving olanzapine 5 to 20 milligrams mg ; day ; for the treatment of schizophreniform disorder. During the fifth week of olanzapine therapy, the patient developed spontaneous whitish discharge from both breasts and reported missing her menstrual period. Her serum prolactin level was 146.55 nanograms milliliter ng ml ; normal range, 1.5 to 19 ng ml.

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